Post Cycle Therapy
Information about products
CHEMICAL NAME: (Z) -2 [4-(1,2-diphenylbut-1-enyl) phenoxy]-N, N-dimethylethanamine
HALF-LIFE: 7-14 hours
Tamoxifen citrate is a non-steroidal anti-estrogen with a wide use in clinical medicine. It belongs to a class of selective estrogen receptor modulators (SERMs), and also works as an estrogen agonist and antagonist. In some tissues, it can act as estrogen, but in others it blocks the effects of estrogen. In the mammary tissue it acts as a potent anti-estrogen, and consequently is commonly used in the treatment of breast cancer.
The main scarecrow among the main side effects of anabolic steroids is gynecomastia, or the very unsightly development of female breast tissue in men. Initially, this may appear as swelling or a small lump under the nipple. If left untreated, it may develop into the hard tissue of gynecomastia, which cannot be removed other than by surgery.
There is a belief that tamoxifen reduces the gains acquired during steroid treatment. This stems precisely from the most important properties of tamoxifen to block estrogen receptors and consequently limit the estrogenic side effects. Anabolic steroids causing the biggest mass and weight gains are those that also have estrogenic side effects, especially excessive water retention (testosterone, methandienone, nandrolone ...).Majority of these gains come from excessive water retention in the muscles and after stopping the treatment, gains disappear as quickly as they appeared. If during the use of the above anabolic steroids you include tamoxifen, the body will retain less water, resulting in a lower weight gain.
USE IN PCT
To suppress the natural production of sex hormones is one of several unwelcome aspects of the use of anabolic steroids. As soon as you stop taking steroids, it can take weeks or even months until the body resumes the natural production of hormones. It should therefore be the main focus of each doping consumer to care for the fastest restoration of natural creation.
The role of antiestrogen tamoxifen is to block the estrogen negative feedback, thereby increasing the level of luteinizing hormone (hormone that stimulates the production of testosterone).
Estrogen levels after stopping the treatment are not actually increased but rather decreased. Any increase in estrogen levels after the end of treatment occurs with restoration of testosterone levels (not before restoring testosterone). But the problem is the imbalance between testosterone and estrogen.
It is important to note that anti-estrogens alone are insufficient to restore normal endogenous testosterone. It is therefore necessary to use a combination of substances in your program for restoring the natural creation of testosterone, most often hCG and antiaromatases (exemestane, anastrozole).
To mitigate estrogenic side effects of anabolic steroids, usually sufficient dose ranges between 10-30mg.
As part of the PCT, tamoxifen is used in an amount of 20 mg daily for 45 days from the start of the program. Together with human chorionic gonadotrophin (hCG), which is taken every other day in an amount 1000ui every other day and exemestane in an amount of 25 mg per day for two weeks.
|Effective prevention against the development of gynecomastia||in some tissues acts as estrogen|
|Relieves excessive water intake||is toxic to the liver|
|Important part of PCT|